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Aloc (Allopurinol)

Aloc is a medication used for gout treatment, provoked by metabolism abnormality with serious affection on joints. Generally, it is used for treating acute attacks of gout, erosive destructive gouty joint disease, uric acid deposits in tissues gouty kidney disease, and uric acid stones. Aloc is used for treating gout caused by excessive levels of uric acid in the blood (hyperuricemia). Hyperuricemia occurs when the body produces more uric acid than it can eliminate. The daily dosage of Aloc is 100-800 mg.

Other names for this medication:
Allopurinol, Zyloprim, Allosig, Allupol, Aloc, Alomaron, Alopron, Aluron, Darinol, Goutex, Llanol, Loric, Prodec, Puribel, Puricos, Purinase, Purinol, Talol, Tipuric, Uriprim, Zurim,

Similar Products:
Cefpodoxim, Clindacne, Flexin, Visacor, Renadinac, Alltop, Omax, Loxitan, Zollocid, Atova, Bicalutamide, Alzo, Overal, Lamotrix, Amoxipenil, Sumamigren, Viranet, Luke, Amohexal, Utinor, Losacor, Quinobiot, Confortid, Tryme, Emflam, Vostar, Daxid, Dulane, Aquazide, Axodin, Dazomet, Spidufen, Gastroplex, Mectizan, Cutaclin, Esidrex, Myclin, Topra, Olmec, Orlistat, Clopivas, Rovartal,

Aloc is used to prevent or lower high uric acid levels in the blood. It is also used to prevent or lower excess uric acid levels caused by cancer medicines or in patients with kidney stones. A high uric acid level can cause gout or gouty arthritis (joint pain and inflammation). Aloc is a xanthine oxidase inhibitor that works by decreasing the uric acid produced by the body.

This medicine is available only with your doctor's prescription.

This product is available in the following dosage forms:

  • Tablet

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Rapid analysis of essential and branched-chain amino acids in nutraceutical products by micellar electrokinetic capillary chromatography. Network Analysis Reveals Similar Transcriptomic Responses to Intrinsic Properties of Carbon Nanomaterials in Vitro and in Vivo.

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We demonstrate that in contrast to the clear dependence of CLL on BCL-2 alone, effective antileukemic activity in the majority of ALL xenografts requires concurrent inhibition of both BCL-2 and BCL-XL We identify BCL-XL expression as a key predictor of poor response to venetoclax and demonstrate that concurrent inhibition of both BCL-2 and BCL-XL results in synergistic killing in the majority of ALL xenografts. The ridge regression best linear unbiased prediction (rr-BLUP) and Bayesian stochastic search variable (BSSV) models presented equivalent abilities regarding predictive processes.

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